As for the mutation bit I've got no idea how likely/possible that would be but, using logic alone, it does not seem to me to be a huge evolutionary leap for the disease to become communicable via coughing / sneezing as the current method of infection is via any sort of bodily fluid.
Does not work like that. When the virus mutates not only one variable changes. It may spread more easily but be less virulent. Or even more virulent, killing the host twice faster and therefore limiting the spread of the disease. If that were that easy we would be surrounded by killer diseases by now, yet this is FAR from being the case.
Hands up everyone posting here that's a medical professional.
No-one? Oh, just me then.
We
are surrounded by killer diseases. They're getting worse every year, though the spread isn't anything like as rapid as Ebola - but that's mostly because Ebola is spread by ignorance and questionable social practices.
In the 1970s, Staphylococcus Aureus mutated to become resistant to the wonder-drug of choice, Meticillin. It's a killer, and it's everywhere but thanks to the efforts of the WHO it's no longer serious and we can treat it. It mainly kills the immunocompromised, the elderly and the very young. It's in every home in the whole world now and the vast majority of people are colonised with it.
We developed new drugs (antibiotics) to deal with it - mostly Vancomycin, Mupirocin and for decolonisation we use things like Triclosan. It's a serious problem for surgical procedures as most people carry it and it plays havoc with wounds. Yet we still deal with it on a daily basis. Thanks to the WHO, you'll be tested for it and cured of it (for the short term, until you go home and become recolonised) prior to anything risky.
Clostridium Difficile is on the rise and that kills people by rotting their intestines. Again, it's a twat to destroy - its spores can survive in 90% ethanol for more than twenty years - but we deal with it using the same Antibiotics as we do for MRSA, Vancomycin.
Just recently we've discovered Carbapenemase-producing Enterobacteriacae - a mutation of low-grade bacteria which is not only resistant to our top-line antibiotics (Carbapenems) but can actually
eat them, if you will. There is no known cure or remedy and it will kill at least ten percent of cases, probably more.
Then there's Vancomycin-resistant Staphylococcus Aureus - a mutation of MRSA that is resistant to all known antibiotics and will almost certainly (according to biochemists at Sheffield's research wing of the University) kill ten percent of the world's population within our lifetimes - and again, there's no cure.
And the most (un)surprising thing is that
nobody is researching new antibiotics for these last two - biochemists have given up on them and are looking now to biological treatments (quorum-sensing looks promising, as does nanotech in current research).
Ebola is worrying, as is SARS and H1N1, because they're viruses. Our arsenal of antivirals is small, and largely ineffective. Most patients in my care are brought through a nasty viral infection not by killing the virus but by managing symptoms - reducing fever, hydration, ventilation etc. A few years ago, someone dropped dead in front of me from Swine Flu - it was horrific to watch but there was nothing we could do; he had waited too long for treatment.
The point is that we've run out of options for Bacteria - they've won the war. Viruses are even worse because we don't have
any effective tools at our disposal and if just one runs out of control we're seriously fucked - it'd be influenza 1918 all over again and the death toll would be massive. That's why the WHO bleat about how serious things might be, and the media unfortunately sensationalise it.
We've been lucky so far, all the flu-variants have been rather mild. Ebola probably will be too, but can we take the chance that it might not be?
One final thought for you all - Humans' evolution has happened over a very, very long timescale as we don't tend to reproduce in binary manner. Bacteria (and to a lesser extent, viruses) evolve very, very fast - so fast you can
see it happen under a microscope. So how likely is a mutation?
Thank fuck Ebola is a virus.
D.